The majority oppressed? On asymmetrical multiculturalism and majority rights

There has been an increase of academic publications that argue in favor of ‘majority rights,’ ‘majority precedence,’ or ‘white identity,’ claiming that the (cultural) interests of majorities in liberal-democratic countries have been ignored due to ‘asymmetrical multiculturalism.’ This article critically examines this academic trend. In particular, we question the claim that liberal-democratic and multicultural theories to date have ignored the importance of the majority (culture). We observe that liberal-democratic and multicultural theory in fact strongly promote and privilege the majority culture, although in ways that do not violate core individual rights and accommodate minorities. In addition, we explore several more empirical issues regarding the claim that the interests of majorities are under threat in liberal-democratic countries today. Among other things, we observe that pro-majority theories tend to work with specific understandings of who embodies the majority. These theories rest on the idea that immigrants and their descendants (may) ‘dilute’ majorities, as they are (culturally) ‘not native.’ As a result, majority rights theorists ‘freeze’ the majority culture claimed to be worth protecting in ways that it, first, neglects ongoing processes of integration and, second, disregards possibilities for social and political change and emancipation, particularly if triggered by immigrant groups. Finally, we wonder why majority rights theories currently seem to resonate. We discuss the possibility that certain pro-diversity voices, such as those who claim that Europe has become superdiverse or who defend multiculturalism, might have (unintentionally) enabled alarmist defenses of majority rights

A method to impregnate wet soil samples, producing high-quality thin sections.

[120.031]Mineral soils with different moisture content and texture were impregnated successfully by the method described. The water present in the soil sample is replaced by acetone and the acetone-saturated sample is impregnated by FitzPatrick's method (1970) (using acetone as thinner of the polyester resin Synolith 544). Shrinkage is minimal and the thin sections are of high quality. It should be possible to correlate thin section data obtained by this method with physical data obtained from undisturbed material. Micromorphologically, a better understanding of the plasmic fabrics should be possible. (Abstract retrieved from CAB Abstracts by CABI’s permission

Colorectal cancer is associated with increased circulating lipopolysaccharide, inflammation and hypercoagulability

Gut dysbiosis contributes to the development of a dysfunctional gut barrier, facilitating the translocation of bacteria and inflammagens, and is implicated in colorectal cancer (CRC) pathogenesis. Such ‘leaky gut’ conditions result in systemic inflammation, of which a hallmark is increased hypercoagulability. Fluorescence antibody confocal microscopy was used to determine circulating levels of lipopolysaccharide (LPS) in control and CRC populations. Here we showed that circulating levels of LPS are significantly elevated in the CRC population. We also showed that markers of inflammation and hypercoagulability are increased in this population. Furthermore, anomalous blood clotting and structural changes in blood components are presented. Importantly, the association between LPS levels, inflammation, and hematological dysfunction was analysed. Statistical regression models were applied to identify markers with strong association with CRC, and to investigate the correlation between markers. A core aim is enhanced biomarker discovery for CRC. We conclude that circulating LPS can promote systemic inflammation and contribute to the development of a pathological coagulation system, with resulting chronic inflammation and an activated coagulation system implicated in tumorigenesis. Blood-based screening tools are an emerging research area of interest for CRC screening. We propose the use of additional (novel) biomarkers to effectively screen for CRC